我在环境中使用边注multicol。\reversemarginpar命令用于触发边注左右边距放置。但\reversemarginpar命令在多列环境中不起作用。
我的要求是边注触发器在包内的左右位置multicol。如何实现要求
平均能量损失
\documentclass[twocolumn]{article}
\usepackage{authblk}
\usepackage{endnotes}
\usepackage{graphics}
\usepackage{multicol}
\usepackage{graphicx}
\usepackage{etoolbox}
\usepackage{marginnote}
% ------------- comment this at the first run -------------
\AtBeginDocument{\theendnotes\clearpage%
}
%----------------------------------------------------------
\begin{document}
\title{Sample document for endnotes}
\author[1,]{Junli Liu}
\author[2]{James Rowe}
\author[2]{Keith Lindsey}
\affil[1]{Integrative Cell Biology Laboratory, School of Biological and Biomedical Sciences, The Bio physical Sciences Institute, Durham University, Durham, UK}
\maketitle
\noindent Patterning\reversemarginpar\endnote{Sample Query Endnote One} in Arabidopsis root development
is coordinated via a localized auxin concentration maximum in the
root tip (Sabatini et al., 1999), requiring the regulated expression
of speciÞc genes. This auxin gradient has been hypothesized to be sink-driven (Friml et al., 2002) and com- putational modeling suggests that auxin efßux carrier activity may be sufÞcient to generate the gradient in the absence of auxin biosynthesis\endnote{Sample Query Endnote Two} in the root (Grieneisen et al., 2007; Wabnik et al., 2010). However, other experimental studies show that local auxin biosynthesis modulates gradient- directed planar polarity in Arabidopsis, and a local source of auxin biosynthesis contributes to auxin gradient homeostasis (Ikeda et al., 2009). Thus genetic studies show that auxin biosynthesis (Ikeda et al., 2009; Normanly, 2010; Zhao, 2010), the AUX1/LAX inßux carriers (Swarup et al., 2005, 2008; Jones et al., 2008; Krupinski and Jonsson, 2010), and the PIN auxin efux carri- ers (Petr‡sek et al., 2006; Grieneisen et al., 2007; Krupinski and Jonsson, 2010; Mironova et al., 2010) all play important roles in the formation of auxin gradients. In addition, experimental evidence shows that, in root devel- opment, hormones and the associated regulatory and target genes form a network, in which relevant genes regulate hormone activ- ities and hormones regulate gene expression. For example, ethy- lene promotes auxin ßux in the root, in a process dependent on the POLARIS (PLS) peptide (Ruzicka et al., 2007; Swarup et al., 2007; Liu et al., 2010a). Furthermore, PIN levels are positively reg- ulated by ethylene and auxin in Arabidopsis roots (Ruzicka et al., 2007). Interestingly, cytokinin can negatively regulate PIN lev- els (Ruzicka et al., 2009), while repressing auxin biosynthesis and promoting ethylene responses (Nordstrom et al., 2004; Chandler, 2009; Liu et al., 2010a). Cytokinin also has the capacity to mod- ulate auxin transport, by transcriptional regulation of the PIN genes (Ruzicka et al., 2009).
\begin{figure}[!t]
\caption{The sample caption\label{fig:1}}\endnote{Sample Query Endnote Three}
\end{figure}
\noindent Patterning in Arabidopsis root development
is coordinated via a localized auxin concentration maximum in the
root tip (Sabatini et al., 1999), requiring the regulated expression
of speciÞc genes. This\endnote{Sample Query Endnote One} auxin gradient has been hypothesized to be
sink-driven (Friml et al., 2002) and com- putational modeling
suggests that auxin efßux carrier activity may be sufÞcient to generate the gradient in the absence of auxin biosynthesis\endnote{Sample Query Endnote Two} in the root (Grieneisen et al., 2007; Wabnik et al., 2010). However, other experimental studies show that local auxin biosynthesis modulates gradient- directed planar polarity in Arabidopsis, and a local source of auxin biosynthesis contributes to auxin gradient homeostasis (Ikeda et al., 2009). Thus genetic studies show that auxin biosynthesis (Ikeda et al., 2009; Normanly, 2010; Zhao, 2010), the AUX1/LAX inßux carriers (Swarup et al., 2005, 2008; Jones et al., 2008; Krupinski and Jonsson, 2010), and the PIN auxin efux carri- ers (Petr‡sek et al., 2006; Grieneisen et al., 2007; Krupinski and Jonsson, 2010; Mironova et al., 2010) all play important roles in the formation of auxin gradients. In addition, experimental evidence shows that, in root devel- opment, hormones and the associated regulatory and target genes form a network, in which relevant genes regulate hormone activ- ities and hormones regulate gene expression. For example, ethy- lene promotes auxin ßux in the root, in a process dependent on the POLARIS (PLS) peptide (Ruzicka et al., 2007; Swarup et al., 2007; Liu et al., 2010a). Furthermore, PIN levels are positively reg- ulated by ethylene and auxin in Arabidopsis roots (Ruzicka et al., 2007). Interestingly, cytokinin can negatively regulate PIN lev- els (Ruzicka et al., 2009), while repressing auxin biosynthesis and promoting ethylene responses (Nordstrom et al., 2004; Chandler, 2009; Liu et al., 2010a). Cytokinin also has the capacity to mod- ulate auxin transport, by transcriptional regulation of the PIN genes (Ruzicka et al., 2009).
\onecolumn
\begin{multicols}{3}
\noindent Patterning in Arabidopsis root development
is coordinated via a localized auxin concentration maximum in the
root tip (Sabatini et al., 1999), requiring the regulated expression
of speciÞc genes. This\endnote{Multi One} auxin gradient has been hypothesized to be
sink-driven (Friml et al., 2002) and com- putational modeling
suggests that auxin efßux carrier activity may be sufÞcient to generate the gradient in the absence of auxin biosynthesis\endnote{Multi Two} in the root (Grieneisen et al., 2007; Wabnik et al., 2010). However, other experimental studies show that local auxin biosynthesis modulates gradient- directed planar polarity in Arabidopsis, and a local source of auxin biosynthesis contributes to auxin gradient homeostasis (Ikeda et al., 2009). Thus genetic studies show that auxin biosynthesis (Ikeda et al., 2009; Normanly, 2010; Zhao, 2010), the AUX1/LAX inßux carriers (Swarup et al., 2005, 2008; Jones et al., 2008; Krupinski and Jonsson, 2010), and the PIN auxin efux carri- ers (Petr‡sek et al., 2006; Grieneisen et al., 2007; Krupinski and Jonsson, 2010; Mironova et al., 2010) all play important roles in the formation of auxin gradients. In addition, experimental evidence shows that, in root devel- opment, hormones and the associated regulatory and target genes form a network, in which relevant genes regulate hormone activ- ities and hormones regulate gene expression. For example, ethy- lene promotes auxin ßux in the root, in a process dependent on the POLARIS (PLS) peptide (Ruzicka et al., 2007; Swarup et al., 2007; Liu et al., 2010a). Furthermore, PIN levels are positively reg- ulated by ethylene and auxin in Arabidopsis roots (Ruzicka et al., 2007). Interestingly, cytokinin can negatively regulate PIN lev- els (Ruzicka et al., 2009), while repressing auxin biosynthesis and promoting ethylene responses (Nordstrom et al., 2004; Chandler, 2009; Liu et al., 2010a). Cytokinin also has the capacity to mod- ulate auxin transport, by transcriptional regulation of the PIN genes (Ruzicka et al., 2009).
\reversemarginpar
\noindent Patterning in Arabidopsis root development
is coordinated via a localized auxin concentration maximum in the
root tip (Sabatini et al., 1999), requiring the regulated expression
of speciÞc genes. This\endnote{Multi Three} auxin gradient has been hypothesized to be
sink-driven (Friml et al., 2002) and com- putational modeling
suggests that auxin efßux carrier activity may be sufÞcient to generate the gradient in the absence of auxin biosynthesis\endnote{Multi Four} in the root (Grieneisen et al., 2007; Wabnik et al., 2010). However, other experimental studies show that local auxin biosynthesis modulates gradient- directed planar polarity in Arabidopsis, and a local source of auxin biosynthesis contributes to auxin gradient homeostasis (Ikeda et al., 2009). Thus genetic studies show that auxin biosynthesis (Ikeda et al., 2009; Normanly, 2010; Zhao, 2010), the AUX1/LAX inßux carriers (Swarup et al., 2005, 2008; Jones et al., 2008; Krupinski and Jonsson, 2010), and the PIN auxin efux carri- ers (Petr‡sek et al., 2006; Grieneisen et al., 2007; Krupinski and Jonsson, 2010; Mironova et al., 2010) all play important roles in the formation of auxin gradients. In addition, experimental evidence shows that, in root devel- opment, hormones and the associated regulatory and target genes form a network, in which relevant genes regulate hormone activ- ities and hormones regulate gene expression. For example, ethy- lene promotes auxin ßux in the root, in a process dependent on the POLARIS (PLS) peptide (Ruzicka et al., 2007; Swarup et al., 2007; Liu et al., 2010a). Furthermore, PIN levels are positively reg- ulated by ethylene and auxin in Arabidopsis roots (Ruzicka et al., 2007). Interestingly, cytokinin can negatively regulate PIN lev- els (Ruzicka et al., 2009), while repressing auxin biosynthesis and promoting ethylene responses (Nordstrom et al., 2004; Chandler, 2009; Liu et al., 2010a). Cytokinin also has the capacity to mod- ulate auxin transport, by transcriptional regulation of the PIN genes (Ruzicka et al., 2009).
\noindent Patterning in Arabidopsis root development
is coordinated via a localized auxin concentration maximum in the
root tip (Sabatini et al., 1999), requiring the regulated expression
of speciÞc genes. This\endnote{Sample Query Endnote One} auxin gradient has been hypothesized to be
sink-driven (Friml et al., 2002) and com- putational modeling
suggests that auxin efßux carrier activity may be sufÞcient to generate the gradient in the absence of auxin biosynthesis\endnote{Sample Query Endnote Two} in the root (Grieneisen et al., 2007; Wabnik et al., 2010). However, other experimental studies show that local auxin biosynthesis modulates gradient- directed planar polarity in Arabidopsis, and a local source of auxin biosynthesis contributes to auxin gradient homeostasis (Ikeda et al., 2009). Thus genetic studies show that auxin biosynthesis (Ikeda et al., 2009; Normanly, 2010; Zhao, 2010), the AUX1/LAX inßux carriers (Swarup et al., 2005, 2008; Jones et al., 2008; Krupinski and Jonsson, 2010), and the PIN auxin efux carri- ers (Petr‡sek et al., 2006; Grieneisen et al., 2007; Krupinski and Jonsson, 2010; Mironova et al., 2010) all play important roles in the formation of auxin gradients. In addition, experimental evidence shows that, in root devel- opment, hormones and the associated regulatory and target genes form a network, in which relevant genes regulate hormone activ- ities and hormones regulate gene expression. For example, ethy- lene promotes auxin ßux in the root, in a process dependent on the POLARIS (PLS) peptide (Ruzicka et al., 2007; Swarup et al., 2007; Liu et al., 2010a). Furthermore, PIN levels are positively reg- ulated by ethylene and auxin in Arabidopsis roots (Ruzicka et al., 2007). Interestingly, cytokinin can negatively regulate PIN lev- els (Ruzicka et al., 2009), while repressing auxin biosynthesis and promoting ethylene responses (Nordstrom et al., 2004; Chandler, 2009; Liu et al., 2010a). Cytokinin also has the capacity to mod- ulate auxin transport, by transcriptional regulation of the PIN genes (Ruzicka et al., 2009).
\end{multicols}
\end{document}
我将在下面提到对默认 endnotes.sty 所做的更改
\def\makeenmark{\fbox{\@makeenmark}}
\def\endnote{\@ifnextchar[\@xendnote{\stepcounter{endnote}%
\protected@xdef\@theenmark{\theendnote}%
\marginnote{\@endnotemark}\@endnotetext}}
下面提到了我的输出和要求：
